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It estimates variance explained directly from single nucleotide polymorphisms and environmental measures. Both methods have great potential to improve public health interventions focusing on risk-based screening that is informed by both genetic and environmental risk factors.
It explains why genetic and environmental factors can have a big impact in some individuals but not in others. Researchers hypothesised environmental factors controlled disease outcome only if a single genetic mutation was present (or vice versa, a genetic mutation to control disease outcome only under certain environmental circumstances).